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Lipid Res. 10,331. Bortz, W. M. (1968). Riochim. Riophys. Acta 152, 619. Brot, N. , Lossow, W. , and Chaikoff, I. L. /. Lipid Res. 3,413. Bucher, N. L. , and Lynen, F. (1960). Riochim. Riophys. Acta 40,49. Byers, S. , and Friedman, M. (1954). ]. Physiol. 179, 79. Chaikoff, I. , Siperstein, M. , Kiyasu, J. , Reinhardt, W. , Dauben, W. , and Eastham, J. F. /. Riol. Chem. 194,407. Chevallier, F. (1967). Advan. Lipid Res. 5,209. Chobanian, A. , and Hollander, W. /. Clin. Invest. 41,1732. Chobanian, A.

When CPIB was fed, there was an immediate decrease in the Cholesterol Turnover in Man 35 slope of the specific activity curve which was compatible with an in­ crease in pool size or with a reduction in the synthesis of new unlabeled cholesterol. Since plasma cholesterol was in fact diminishing, the findings were more likely to have b e e n due to inhibition of choles­ terol synthesis. Cholesterol catabolism is also stimulated by CPIB. By contrast, the administration of ethinyl estradiol produced an increase in the slope of the specific activity curve which suggested a de­ creasing pool or an increase in catabolic rate and cholesterol turnover.

Clin. Res. 17,395. , and Ahrens, E. , Jr. (1965)J. Clin. Invest. 44,1482. Srere, P. , Chaikoff, I. , Treitman, S. , and Burstein, L. S. (1950). J. Biol. Chem. 182,629. , and Portman, O. W. (1964). Arch. Biochem. Biophys. 107,341. , and Law, M. D. (1966). Arch. Biochem. Biophys. 113,143. , and Law, M. D. (1968). Proc Soc. Exp. Biol. Med. 129,363. , and Treadwell, C. R. (1953). Proc. Soc. Exp. Biol. Med. 84,417. , Law, M. , and Treadwell, C. R. (1964). Arch. Biochem. Biophys. 104,128. , and Borgstrom, B.

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