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Lipid Res. 10,331. Bortz, W. M. (1968). Riochim. Riophys. Acta 152, 619. Brot, N. , Lossow, W. , and Chaikoff, I. L. /. Lipid Res. 3,413. Bucher, N. L. , and Lynen, F. (1960). Riochim. Riophys. Acta 40,49. Byers, S. , and Friedman, M. (1954). ]. Physiol. 179, 79. Chaikoff, I. , Siperstein, M. , Kiyasu, J. , Reinhardt, W. , Dauben, W. , and Eastham, J. F. /. Riol. Chem. 194,407. Chevallier, F. (1967). Advan. Lipid Res. 5,209. Chobanian, A. , and Hollander, W. /. Clin. Invest. 41,1732. Chobanian, A.
When CPIB was fed, there was an immediate decrease in the Cholesterol Turnover in Man 35 slope of the specific activity curve which was compatible with an in crease in pool size or with a reduction in the synthesis of new unlabeled cholesterol. Since plasma cholesterol was in fact diminishing, the findings were more likely to have b e e n due to inhibition of choles terol synthesis. Cholesterol catabolism is also stimulated by CPIB. By contrast, the administration of ethinyl estradiol produced an increase in the slope of the specific activity curve which suggested a de creasing pool or an increase in catabolic rate and cholesterol turnover.
Clin. Res. 17,395. , and Ahrens, E. , Jr. (1965)J. Clin. Invest. 44,1482. Srere, P. , Chaikoff, I. , Treitman, S. , and Burstein, L. S. (1950). J. Biol. Chem. 182,629. , and Portman, O. W. (1964). Arch. Biochem. Biophys. 107,341. , and Law, M. D. (1966). Arch. Biochem. Biophys. 113,143. , and Law, M. D. (1968). Proc Soc. Exp. Biol. Med. 129,363. , and Treadwell, C. R. (1953). Proc. Soc. Exp. Biol. Med. 84,417. , Law, M. , and Treadwell, C. R. (1964). Arch. Biochem. Biophys. 104,128. , and Borgstrom, B.